Is “fatty liver + high insulin” caused by obesity? The fundamental problem is hormones

Have you ever had this experience: your physical examination report shows fatty liver and high fasting insulin, but your weight is not too exaggerated? Or you are trying to lose weight, controlling your diet and exercising hard every day, but your weight loss is slow and you can't lose weight ? Many people think that these problems are just because of " eating too much and exercising too little " , but the truth is - weight is just a symptom, and the real source lies in the imbalance of hormones and metabolic mechanisms.

Fatty liver + high insulin are not the result of being fat , but the body's metabolic system has already sounded the alarm . To truly reverse it, starting with hormones and repairing metabolism is the key .

 

1. The truth behind “ fatty liver + high insulin ” : it is not because of obesity that metabolism is poor, but poor metabolism that causes obesity

📌 Analysis of common mechanisms:

•       Insulin resistance : Insulin secretion is normal or even excessive, but cells are " numb " to it, glucose cannot be fully utilized, and is instead synthesized into fat for storage

•       Fat accumulation in the liver : Free fatty acids continue to enter the liver, forming non-alcoholic fatty liver disease ( NAFLD ), further aggravating insulin resistance

•       Chronic inflammation and hormone imbalance : The imbalance of cortisol, leptin, ghrelin, etc. keeps the body in " energy storage mode " , making it easy to gain weight no matter how much you eat, and it is difficult to lose weight quickly no matter how much you lose weight.

✅ Many people already have these hormone and metabolic problems before their BMI exceeds the standard , especially those with abdominal obesity and high visceral fat.

 

2. Why are traditional weight loss methods almost ineffective for this type of people?

•       Dieting → low calorie intake, but basal metabolism further decreases, the body is more energy-efficient

•       Exercise → It is difficult to reverse insulin resistance, and the consumption efficiency is always limited

•       Unrepaired metabolism → weight loss → rebound → more severe metabolic disorder cycle

📌 It’s not that you can’t lose weight, but that your “ hormone system has not been awakened ” and your body is still in the “ fat storage ” mode.

 

3. Tipoline: Restarting the metabolic system by starting with the hormone mechanism

Element	Mechanism of action	Significance for fatty liver and hyperinsulinemia
GLP-1 agonists	Suppress appetite, delay gastric emptying, and promote insulin secretion	Stabilize postprandial blood sugar and reduce the burden on pancreatic β cells
GIP agonists	Improve insulin sensitivity and promote fat metabolism	Reverse insulin resistance and reduce visceral fat accumulation

📌 Telportide does not just reduce weight, but also rebuilds hormones:

•       Reduce liver fat synthesis

•       Improves muscle responsiveness to insulin

•       Let the body switch from " energy storage mode " to " energy consumption mode "

✅ This is the real way to solve " high insulin + fatty liver " from the source.

 

4. Clinical research data: not only weight loss, but also improvement of metabolic abnormalities

📌 SURMOUNT-1 study ( 72 weeks) found:

•       Average weight loss: 22.5% ; waist circumference reduced by 19cm

•       Insulin resistance index ( HOMA-IR ) decreased significantly

•       The average liver fat content decreased by more than 30% (imaging test)

📌 The SURPASS-3 study (patients with diabetes + fatty liver) showed:

•       Tipoline significantly improves liver function indicators such as ALT and AST

•       Fasting insulin lowering effect is better than semaglutide

 

5. Who is suitable for considering telotropin to improve hormone metabolism?

📌 Recommended for:

•       Physical examination shows fatty liver and elevated fasting insulin ( >15μU/mL )

•       BMI ≥ 27 , with large waist circumference and high visceral fat

•       Failed to lose weight many times, severe rebound, suspected insulin resistance or prediabetes

•       Those with a family history of diabetes, hyperlipidemia, polycystic ovary syndrome, or fatty liver disease

📌 Recommended use:

•       Initial dose: 2.5 mg/ week, gradually increased

•       Dietary recommendations: high protein, low refined carbohydrates

•       Combined with monitoring of insulin, fasting blood sugar, liver function and fatty liver imaging indicators

 What you need is not a diet pill, but a " metabolic repair program "

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